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Low fat, high fibre childhood diet 'prevents metabolic syndrome'

Thursday 27th October 2011

A childhood diet that is lower in fat and higher in fibre could lower the risk for chronic diseases in the long-term, scientists have said.

Research published in the Journal of Clinical Endocrinology and Metabolism revealed that childhood behavioural intervention to lower dietary intake of total fat and saturated fat and increase consumption of nutritious foodstuffs could lower the risk of heart conditions.

When the children were re-evaluated in young adulthood it was found that the ones who had changed their diets had significantly lower fasting plasma glucose levels, as well as lower systolic blood pressure.

A Western dietary pattern high in total fat and saturated fatty acids and refined grains is linked to an increased risk of metabolic syndrome, a group of metabolic abnormalities which include obesity, low levels of high-density

lipoprotein cholesterol, higher levels of triglycerides and blood glucose, and increased blood pressure.

This research indicates that small reductions in total fat and saturated fat, alongside increased dietary fibre during childhood and adolescence could have a beneficial effect later in life.

Lead author Joanne Dorgan, of Fox Chase Cancer Center in Philadelphia, said: "This research is important because it suggests that modest reductions in total fat and saturated fat intake and increased consumption of dietary fibre

during childhood and adolescence may have beneficial effects later in life by decreasing risk of chronic diseases such as diabetes and heart disease."

In other news, a blood test could be used to measure the danger of heart disease in smokers, according to a study published in journal Arteriosclerosis, Thrombosis and Vascular Biology.

Researchers were able to work out the amount of pulmonary surfactant B (SP-B), a protein which is found in damaged lung cells, in the blood.

Participants who had higher levels of SP-B were also observed to have more build-up of dangerous plaque in the aorta.

Written by James PuckleADNFCR-1780-ID-800778765-ADNFCR

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